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Topic: COVID-19 - NOT PRESENTING as VIRAL INDUCED PNEUMONIA in NYC HOSPITALS  (Read 1095 times)

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I think that this video, literally from "ground zero" -- in more ways than one-- is VERY, VERY important.  At the time of this writing, NYC is still the epicenter of COVID-19, with the next wave of "epicenters". following about seven to ten days behind (or so they say).

Pay careful attention to what the ER doctor is describing, and contrast that to the Earthcures recommended treatment strategy.

This doctor lends a great deal of credence to the idea that some strange thick solidifying mucus may be a significant contributing factor to the cause of death, just like the autopsy doctors hinted at in China.

Those ER and ICU doctors and nurses are so out their league it is frightening... even terrifying.

Since the docs from China also indicated that it would take up to 5 days to break down that mucus once patients were admitted to ICU, it is CRITICAL that this be a main priority in order to avoid what would otherwise become the worse ordeal anyone would experience.


UPDATE:  The doctor that shot this video eventually resigned over the hospital's policy on ventilator use.

Here is a "newer" account of this same issue, given to a friend by a nurse working at the same hospital:

The doctor that made the original video questioning how ventilators were being used, eventually resigned over it.

Here is another perspective, I believe from the same hospital.  This is not easy to watch, and I believe that it is 100% authentic:

https://www.dailymail.co.uk/news/article-8262351/Nurse-New-York-claims-city-killing-COVID-19-patients-putting-ventilators.html

If all of this isn't enough to convince a person to take care of one's health and act quickly to AVOID going into the hospital, I don't know what would!

On a related "aside", I read a report from a nurse working in ICU in a California hospital who shared that they have completed changed their ventilator use based on the experience reported coming out of New York.

But, sadly, while CPAP and BiPAP both have proven to be HIGHLY effective and MUCH better than ventilators, many hospitals will not use them because they also risk spreading the virus in aerosol form.

Here is another situation where us not being properly prepared is costing people a great deal of pain, misery and even death.  If hospitals would put in place a policy of converting ICU rooms to negative air flow, properly designed rooms with UV air filtration as well, this would not be an issue.

Would this be expensive?  Comparatively, yes.  But, people are not given the choice, and people are not told the truth at all.

They are simply frightened into accepting forced ventilation as their only treatment option.  Would there be situations where a ventilator would HAVE to be used?  Absolutely most certainly... but not in the same manner as they are being used now, and not as frequently.

I believe that most hospital policies prioritize forced ventilation due to all of the initial reports coming out of Italy, where doctors reported that patient outcomes were GREATLY improved if forced ventilation was used for EARLY intervention.  I truly wonder what the disconnect has been between observations in places like Italy, as composed to places like New York intensive care units.

Also, doctors are getting a 100%+ increase in patient blood oxygen levels (those with low levels due to COVID-19) by using warmed and humidified oxygen via high flow O2 therapy using a simple, non-evasive nose cannulas. 

This can even be tried at home with a good quality oxygen concentrator.  Most have optional humidifier feeds in-line. 

If you have a simple oxygen saturation meter (the "cheap" fingertip monitors that measure blood oxygen saturation and current heart rate), you can do this with no risk.  Everyone should be testing their O2 saturation levels anyway, if at all possible.

The operative idea would be to monitor your O2 levels, and then add the O2 if oxygen saturation drops to below ~90% - 94% (you should have a normal baseline established so that you know what YOUR "normal" O2 saturation level is...before any respiratory distress).

This link documents how UChicago Medicine is using this therapy with great success, rather than turning to ventilators:

https://www.uchicagomedicine.org/forefront/coronavirus-disease-covid-19/uchicago-medicine-doctors-see-truly-remarkable-success-using-ventilator-alternatives-to-treat-covid19

They are not the only medical professionals trying to opt out of early ventilator intervention.  There are a LOT of medical professionals who aren't willing to simply apply failing protocols that they know will likely lead to a high patient mortality rate.  Some are even testing out breathing ozonated saline solution to address the infection:

https://www.facebook.com/H2INJECTION/videos/825101194649957/

Also, to read all of our current material on COVID-19, don't forget to visit our landing page:

https://www.earthcures.org/forum/index.php/topic,184.0.html

Continue reading below for the original post responses that were added as we learned more information.  Most of it was posted prior to the "new" updates shown above.
« Last Edit: May 03, 2020, 10:11:51 AM by Jason »
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...some doctors suspect that while the virus is attaching to ACE2 cell receptors in the lungs, that the virus is breaking hemoglobin down to use something like iron as a food source.  This is resulting in the destruction of hemoglobin, which can eventually cause oxygen starvation (hemoglobin is what carries oxygen throughout the body to cells).

This means that it becomes extremely important to cut down viral counts and prevent virus replication as early as possible.  Early intervention is critical.


Furthermore, based on these findings, some nurses/doctors in ICU are opting OUT of using ventilators, in favor of using physical poses used by respiratory therapists (such as lying patients on the stomach).  This has been reported to increase blood oxygenation levels by ~10%, and without the risk of lung tissue damaged caused by too much pressure (from the use of forced respiration via ventilators).

https://futurism.com/neoscope/rolling-coronavirus-patients-stomachs-saving-lives

It is very easy to look up OTHER physical poses used by respiratory therapists as well; some may be even more efficient.

While this is proving to be beneficial for patients experiencing reduced blood oxygenation due to lung inflammation, poses designed to improve lung efficiency are certainly ALSO beneficial when individuals are suffering from things like pneumonia, where a fluid/mucus build up may be responsible for breathing issues.

Other "theories" by medical researchers abound.  One interesting theory is that-- like with "regular" influenza pneumonia-- that an opportunistic bacterial infection is responsible for attacking hemoglobin.  Perhaps this is why the original anti-malaria drug protocol also included the use of powerful antibiotics.
« Last Edit: May 03, 2020, 10:07:34 AM by Jason »
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...Silent hypoxia, via a "different type of pneumonia".

This checks many of the boxes I've been staring it for the past few weeks, but not all of them (please see the link below).

This does match the autopsy reports I heard about from China; the fluid build up is slow and not excessive, to the point where people aren't even aware of it.  It starts to solidify before a person has the chance to take any action.  It does not seem to be an excessive amount of fluid/mucus, either.

So, which is it?  Is it COVID-19 breaking down hemoglobin that is causing oxygen starvation (using iron as a food source), or is the oxygen starvation caused by a thin layer of fluid that starts to solidify blocking airways along with inflammation and fibrotic tissue?

It may be a combination of all of the above.

While not definitive, it's valuable information to keep in mind.  Plus, there is nothing wrong with checking your O2 blood saturation levels anyway!

https://www.nytimes.com/2020/04/20/opinion/coronavirus-testing-pneumonia.html?smid=fb-share

No matter the cause, it really does not change the treatment strategy...  EXCEPT for, perhaps, individuals being more diligent and checking their blood oxygen saturation on a daily basis.  It is easy to do, and there is no down side (aside from paranoia).

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...not directly related to the pneumonia issue, but this is as good of a space to discuss it as any...

A very limited study (3 patients) done by a doctor suggests that the virus doesn't just attack the lungs, primarily, but the lining of blood vessels throughout the body.  This lends credence to the idea that those with any form of cardiovascular disease are at far greater risk for death.

I would be interested to see what further research uncovers, since this contradicts previously posted research (there is a lot of that going around lately!!!).

In particular, I'm referring to blood analysis that shows less than 1% of the virus exists in the blood stream.

SO, does the virus attached to the ACE2 cell receptors of the lungs, replicate there, and chase hemoglobin around the body?

Luckily, this doesn't change the fundamental treatment strategy, since stopping viral replication is central to beating COVID-19.

https://www.nytimes.com/2020/04/20/opinion/coronavirus-testing-pneumonia.html

It does, however, strengthen the case for using systemic enzymes for prevention and treatment (see the "Rumor Mill" page for further information about the Dr. Wong's suggestion of using systemic enzymes for COVID-19).
« Last Edit: April 21, 2020, 06:09:18 PM by Jason »
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I've been puzzled for weeks at the amount of contradictory data being presented from all over the place.  It has been very puzzling, including differences in how COVID-19 expresses as a pathogenic organism.

...this rather disturbing report from one of China's most qualified COVID-19 researchers documents the mutating ability of the virus, including some strains that produce more than 270 TIMES the amount of viral loads.  This would go a very long way to help answer the puzzling contradictions of data out there.

Furthermore, the scientist concludes:  "If there is a discovery that overturns the prevailing perception, don’t be surprised.”

This may help explain why this virus is so lethal in some locations, and mild in others.

https://www.scmp.com/news/china/science/article/3080771/coronavirus-mutations-affect-deadliness-strains-chinese-study

Even up to last week, "government" experts had touted, on mainstream media (both The BBC and CNN, if I remember correctly), that this virus could not and will not mutate.  We've known this wasn't true for almost two months, at the time of this updated writing.

Well, now Los Alamos Lab in New Mexico has confirmed the research we posted (above) out of China.  There is a much more virulent, dominant strain of Sars-CoV-2 emerging.  This reinforces one the reasons we believe that there is a difference in reported cases in different demographic regions.

Unfortunately,  arm chair "pundits", both online and in other media, are still cherry picking data sets which support their "political" position on the nature of this pandemic.

Here is a link to the Los Alamos data:

https://onezero.medium.com/swiss-scientists-have-recreated-the-coronavirus-in-a-lab-d12816bfdbe3
« Last Edit: May 06, 2020, 12:17:00 PM by Jason »
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As the COVID-19 "mystery" continues, more and more startling-- and contradictory-- observations are being made.

Are the doctors that dealt with all of the early cases of COVID-19 inept?  Is the disease pathology changing and mutating?  Do different people experience completely different pathologies?

If you are confused by the recent reports, including the new article published in the Washington Times ( https://www.washingtonpost.com/health/2020/04/22/coronavirus-blood-clots/), you are not alone.

I strongly recommend studying all of the information above, in order.  Take the time to watch any videos, or read the associated articles.

The most recent "shocking" article in the Washington Post documents that many doctors are noticing a "new" and terrifying attribute to the pathology of COVID-19 in patients.  Even when patients are being put on anti-coagulants, they are suddenly dying of complications due to blood clots.

To quote the article:

"Increasingly, doctors also are reporting bizarre, unsettling cases that don’t seem to follow any of the textbooks they’ve trained on. They describe patients with startlingly low oxygen levels — so low that they would normally be unconscious or near death — talking and swiping on their phones. Asymptomatic pregnant women suddenly in cardiac arrest. Patients who by all conventional measures seem to have mild disease deteriorating within minutes and dying at home.

With no clear patterns in terms of age or chronic conditions, some scientists hypothesize that at least some of these abnormalities may be explained by severe changes in patients’ blood."

All of these new reports that starkly contrast older reports... simply highlight and underscore the need to treat this infection in the correct manner.

I've had people tell me that they've discovered that the disease is now a blood disease rather than a respiratory one.  They seem to now be looking for ways to treat the blood and blood vessels.

However, this is NOT the correct focus.  Until data changes that documents that the replication of the virus has switched from using its spike protein to attach to the ACE2 cell receptors... to some other means of replication...  the battle against COVID-19 is still either won or lost at the lungs.

Thus far, it appears that the virus works its way into the lungs (from the sinuses and/or the throat), and then attaches to cells with ACE2 receptors.  After a period of around 3-5 days, the virus particle can then hijack the cell in order to replicate.  With some strains, the replication is much less than other strains.  As documented in the above thread, some strains can be hundreds of times more "virulent", making them potentially hundreds of times more deadly.

Now, once a copy of the virus has been completed, the "new" virus particle will attempt to bind to another ACE2 inhibitor (this attachment is done by contact, like a key "blindly" trying to find the proper key hole).  If it does attach to a new cell, this replication strategy is repeated.

Now, all the while, it appears that the virus is breaking down hemoglobin for a source of energy.  Some scientists have theorized that it is after the iron content.  Either way, this process destroys the hemoglobin, whereby it can ****no longer carry oxygen****.

Those new viral particles that fail to attach to a new cell via its ACE2 inhibitor then start to circulate in the blood stream.  Since this is a brand new threat to the body, the body is blindsided, caught completely off guard.  Eventually, it will begin to mount a immune system response.  The risk for a cytokine storm is very real, with no real way, as of yet, to predict who this will happen to.

When cardiovascular disease is involved, the risk of death is much more prevalent.  The same can be said with things like obesity and diabetes.

While the virus is not able to replicate (not being attached to a suitable host cell), it can and will still wreak havoc throughout the cardiovascular system (inflammatory response, fibrotic tissue formation, immune system over-response).  This includes newer observations of severe damage to blood vessels (the blood vessel walls). 

As the copies of the virii circulate throughout the blood stream, they can also cause damage to organs.  It is logical to assume that the virus can cause damage to any system that utilizes blood in primary circulation, which of course includes the liver and kidneys.

Here, we SUSPECT that a person's risk increases in direct association to how great the initial (or repeated) exposure is/was.  Why?  Because the more virus the body has to deal with initially,  the more copies it will be blind sided with when they start to replicate.  The progression is geometric.  So, the healthy doctors that have been dying who never had adequate protection were likely overwhelmed by the virus due to constant/repeated exposure.

No matter the case, the answer doesn't lie in chasing symptoms, the answer still lies in preventing the virus from replicating, and that replication is happening mainly at the lungs (although there are reports of digestive system complaints, even as a first symptom, suggesting that the virus might be attaching to ACE2 cell receptors in the gut).

Protecting the cardiovascular system would be important, but secondary.  Interested readers should carefully explore the "Rumor Mill" post threads, as people such as Dr. Wong suggest that systemic enzymes may have an important role in helping the body combat fibrotic tissue and blood clotting.
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Hi Jason.

There are ACE2 receptors on multiple,..different tissues in the body.
so that means it could (potentially) affect other organs in the body. (like the heart)

'Maybe' (just speculating here) the lungs are hit heard because, its primarily getting spread via oral means & the lungs are the first organ that is encountered.
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You make a great point, and upon a bit further research.. :

"The expression and distribution of the ACE2 in human body may indicate the potential infection routes of 2019-nCoV. Through the developed single-cell RNA sequencing (scRNA-Seq) technique and single-cell transcriptomes based on the public database, researchers analyzed the ACE2 RNA expression profile at single-cell resolution. High ACE2 expression was identified in type II alveolar cells (AT2) of lung, esophagus upper and stratified epithelial cells, absorptive enterocytes from ileum and colon, cholangiocytes, myocardial cells, kidney proximal tubule cells, and bladder urothelial cells10. These findings indicated that those organs with high ACE2-expressing cells should be considered as potential high risk for 2019-nCoV infection..."

It seems I was too focused on the early reports of the pathogenesis from China, Italy, France and Spain... and initial work reported out of New York City.

Now that blood clots are implicated in many deaths (where before all reports of mortality were oxygen/respiratory related), it might be a good idea to expand the thinking.
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There is a lot of news going around, what with the varied symptoms...and not to mention the conspiratorial end of things.

there might be more than 1 strain going around (like you mentioned/quoted in an earlier post) which some hypothize may be responsible for the more aggressive effects in places like Italy

Quote
..I've had people tell me that they've discovered that the disease is now a blood disease rather than a respiratory one. They seem to now be looking for ways to treat the blood and blood vessels ..
..this maybe because of stuff being released/leaked into the blood from the damage being caused in cells, and causing secondary symptoms  ? - the excess cytokines affect the blood vessels too & cause leakage of liquid into the surrounding area.

clots
I had come across some info that said the cytokines being released into the blood stream & inflammation, can affect the pro-coagulant & anticoagulant activity..with an Increase in pro-coagulants,.. which might be causing the clotting (this in turn can cause other effects from an excess of pro-coagulant proteins being used up in these clots)

HIV Genes
this virus is supposed to have 3-4 hiv genes - not sure what if any implications that has.
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« Last Edit: April 24, 2020, 08:50:53 PM by Rainstorm »